Skip to main navigation menu Skip to main content Skip to site footer
×
Español (España) | English
Editorial
Home
Indexing
Review

Role of oxidative stress and therapeutic options in SARS-COV-2 infection

By
Ivan Ronaldo Quilambaqui Brito ,
Ivan Ronaldo Quilambaqui Brito

Universidad Católica de Cuenca, Carrera de Medicina. Cuenca, Ecuador

Search this author on:

PubMed | Google Scholar
Danilo Gustavo Muñoz Palomeque ,
Danilo Gustavo Muñoz Palomeque

Universidad Católica de Cuenca, Carrera de Medicina. Cuenca, Ecuador

Search this author on:

PubMed | Google Scholar

Abstract

Introduction: Elevated levels of oxidative stress in patients with SARS-CoV-2 infection generate tissue damage, causing organ dysfunction and generating a suitable environment for viral replication.
Aim: To describe the mechanisms by which oxidative stress is generated in patients with Sars-Cov-2 and its therapeutic options.
Methodology: A non-experimental and narrative study of bibliographic review type was carried out, data will be collected from original articles in indexed journals using the PubMed database.
Results: Patients with SARS-CoV-2 infection present elevated levels of oxidative stress, on the contrary, the levels of antioxidant agents are depleted, increasing the degree of oxidative stress to a greater extent. The use of N-acetylcysteine in a COVID-19 positive patient is a subject under discussion since, although there are inconsistencies in its degree of efficacy, no adverse effects of any kind have been observed.
Conclusions: there are antioxidant therapeutic options under study, however, despite having a high safety profile, their efficacy in the treatment of COVID-19 is still unproven.

How to Cite

1.
Quilambaqui Brito IR, Muñoz Palomeque DG. Role of oxidative stress and therapeutic options in SARS-COV-2 infection. Salud, Ciencia y Tecnología [Internet]. 2023 Mar. 17 [cited 2024 Apr. 25];3:239. Available from: https://revista.saludcyt.ar/ojs/index.php/sct/article/view/239

The article is distributed under the Creative Commons Attribution 4.0 License. Unless otherwise stated, associated published material is distributed under the same licence.

Article metrics

Google scholar: See link

Metrics

Metrics Loading ...

The statements, opinions and data contained in the journal are solely those of the individual authors and contributors and not of the publisher and the editor(s). We stay neutral with regard to jurisdictional claims in published maps and institutional affiliations.